6
DNA end joining becomes less efficient and more error-prone during cellular senescence.pdf
Accumulation of somatic mutations is thought to contribute to the aging process. Genomic instability has been shown to increase during aging, suggesting an aberrant function of DNA double-strand break (DSB) repair. Surprisingly, DSB repair has not been examined with respect to cellular senescence. Therefore, we have studied the ability of young, presenescent, and senescent normal human fibroblas..
142
MicroRNAs regulate de novo DNA methylation and histone mRNA 3' end formation in mammalian cells.pdf
MicroRNAs regulate de novo DNA methylation and histone mRNA 3’ end formation in mammalian cells
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On RNA 3´-end Oligouridylation(DNA寡聚尿苷).pdf
On RNA 3´-end Oligouridylation(DNA寡聚尿苷)
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Non-Homologous End Joining and Homology Directed DNA Repair Frequency of Double-Stranded Breaks Introduced by Genome Editing Reagents.pdf
RESEARCHARTICLENon-Homologous End Joining HomologyDirectedDNA Repair Frequency Double-StrandedBreaks
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WRN Exonuclease Structure, Molecular Mechanism, and DNA End Processing Role(WRN核酸外切酶的结构,分子机制和DNA末端加工作用).pdf
WRN Exonuclease Structure, Molecular Mechanism, and DNA End Processing Role(WRN核酸外切酶的结构,分子机制和DNA末端加工作用)
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Chapter One - Classical and Alternative End-Joining Pathways for Repair of Lymphocyte-Specific and General DNA Double-Strand Breaks.pdf
Chapter One - Classical and Alternative End-Joining Pathways for Repair of Lymphocyte-Specific and General DNA Double-Strand Breaks
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DNA-PK facilitates piggyBac transposition by promoting paired-end complex formation.pdf
DNA–PKfacilitates piggyBac transposition bypromoting paired-end complex formationYan Jina,1, Yaohui

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